Wednesday 11 September 2013

Max Business Overview


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Max Science Overview

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Max Opportunity

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Dr. Keller and MaxGXL

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Oral Accelerator Improves Hormone Values & Inflammation - Robert H. Kel...


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Dr. Jeanette Roberts speaks of her work with Dr. Herbert T. Nagasawa.


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Dr. Robert Keller talks about Glutathione

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Meet My Partners

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Meet the Doctor Who is Changing My Health

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What is Oxidative Stress?

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Dr. Jeanette Roberts speaks of her work with Dr. Herbert T. Nagasawa.

At 57 Dr. Luis Fuentes is competing with cyclists half his age.

Chad Brown former NFL All Pro player

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Art Sellinger owner of Long Drivers of America

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Bill Gillespie, Five World Records in One Day.

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Bobby Wilson, ESPN Long Drive Champion loves Max

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Evander Holyfield working out like he did 20 years ago..


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Maria Zanandrea, three time Olympian found new energy and stamina.

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David Wells, Marathon runner and Triathlete ran faster on the 16th mile ...


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Jim Phipps is still going strong at 72.


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Ron Brand, major league scout for the New York Yankee, and former MLB pl...


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Henry and Teresa Kramer found better health through Max International pr...


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Frankie Davis shares her experience with Max NFuze and Cellgevity


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Wanda Miller's RiboCeine™ Experience


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Rhonda Dasher


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Chuck and Gena Norris Join Max International

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Nancy Rose Body Builder reports her experience with Max International

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Dr. Charles Mabray explains the benefits of glutathione enhancement.


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Matt Murski, CrossFit Trainer and Competitor

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Dr. Don Colbert talks about glutathione


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Pesticides & Toxins in your Food

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Toxins In Cosmetics / Educational Video

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Max Training Video - Inviting


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Max Training Video - Tools Work!

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The Doctors' Choice Complete Interview

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Max Pay Plan

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Binary Compensation Plan Building Strategy


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Placement Strategy Training Video

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Build to Bronze Training Video


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Max Training Video - Fast Track

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Visible Solutions™ by Max International®, Skin Care Improvements You Can...


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Max International Preview

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Chris Mullin, former NBA Hall of Fame player


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MaxGXL TV Episode 2 (playlist)


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MaxGXL TV Episode 2 (playlist)


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MaxGXL TV Episode 2 (playlist)


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Bobby Wilson (Pro Golfer) Uses MaxGXL - Max International Interview

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Evander Holyfield (Pro Boxer) Uses MaxGXL - Max International Interview


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Max International - Interview with leading Neurologist Dr. Rex Anderson


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Greg Norman (Golfing Legend, International Business Icon)

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Greg Norman, the golfing legend BEFORE Tiger Woods loves our glutathione...

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Dr Don Colbert discusses the importance of glutathione and recommends CE...

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what is glutathione and what is maxgxl video

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Dr Oz on Glutathione - Master Antioxidant!


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Michael Firth Interviews Bryce McGain for The MAX 90 DAY Challenge

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Michael Firth Interviews Georgie Wakim

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Michael Firth interviews Jo Rasche for The MAX 90 DAY Challenge


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Autism and the Role of Glutathione

Autism and the Role of Glutathione

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The incidence of autism spectrum disorders (ASD) has increased dramatically over recent years. In developed countries the incidence has increased by over 600% in the last twenty years.

Other developmental and behavioural disorders like attention deficit disorders (ADD) or attention deficit hyperactivity disorders (ADHD) have also increased up to 1 out of every 6 children in the U.S.

The Mercury Connection

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Mercury is a potent neurotoxin. Worldwide exposure to mercury has increased significantly through fish, industrial sources, vaccinations (containing thimerosal preservative) and dental amalgam fillings. Scientists have found that autistic children may have been exposed to higher amounts of mercury during pregnancy. Glutathione is able to detoxify mercury and other heavy metals. Glutathione levels are known to be depleted in autistic children. 

Glutathione treatment offers hope for autism

It is known that glutathione levels are depleted in autism. Intravenous glutathione supplementation has shown promising results but the treatment is expensive and time consuming, requiring twice weekly infusions.  
The challenge for doctors has been to develop an effective oral supplement, since glutathione is a fragile molecule that is quickly broken down in the digestive system if taken by mouth. 

Following many years of scientific research, effective oral supplements to increase
systemic glutathione levels are now available.  The clinically proven oral supplements 
MaxGXL,MaxOne and Cellgevity provide the cells with the essential precursors that 
they require to make glutathione. These advanced nutritional supplements are ideal for
 supporting brain function, detoxifying cells, improving mental clarity, improving sleep and
 for optimising overall health. 

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Detoxification Process of the Liver







All Max products are composed of powerful antioxidants that  supports glutathione's  directly and indirectly with its protective role in detoxification process of the liver.


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Toxins in Medications

Toxins, poisons and chemicals can cause peripheral neuropathy. This can happen through drug or chemical abuse or through exposure to industrial chemicals in the workplace or in the environment (after either limited or long-term exposure). Common causes include: exposure to lead, mercury, arsenic and thalium. Some organic insecticides and solvents can result in neuropathies. Sniffing glue or other toxic compounds can also cause peripheral neuropathy. Certain herbal medicines, especially Chinese herbal medicines, are particularly rich in mercury and arsenic and taking them can lead to peripheral neuropathy.
Because patients may have subtle pain or weakness, it may be difficult to arrive at a specific diagnosis of toxic neuropathy.

SYMPTOMS AND SIGNS

(Not all symptoms and signs may be present.)
In feet:
  • Pain
  • Tingling
  • Numbness
Other symptoms:
  • Weakness
  • Difficulty walking

Specific drug or toxin

Acetaminophen


Acetaminophen (3D structure) overdose is the most common cause of drug-induced liver disease
Acetaminophen (paracetamol, also known by the brand name Tylenol and Panadol) is usually well tolerated in prescribed dose, but overdose is the most common cause of drug-induced liver disease and acute liver failure worldwide.Damage to the liver is not due to the drug itself but to a toxic metabolite (N-acetyl-p-benzoquinone imine NAPQI, or NABQI) produced by cytochrome P-450 enzymes in the liver. In normal circumstances, this metabolite is detoxified by conjugating with glutathione in phase 2 reaction. In an overdose, a large amount of NAPQI is generated, which overwhelms the detoxification process and leads to liver cell damage. Nitric oxide also plays a role in inducing toxicity.The risk of liver injury is influenced by several factors including the dose ingested, concurrent alcohol or other drug intake, interval between ingestion and antidote, etc. The dose toxic to the liver is quite variable from person to person and is smaller in chronic alcoholics.Measurement of blood level is important in assessing prognosis, higher levels predicting a worse prognosis. Administration of Acetylcysteine, a precursor of glutathione, can limit the severity of the liver damage by capturing the toxic NAPQI. Those that develop acute liver failure can still recover spontaneously, but may require transplantation if poor prognostic signs such as encephalopathy or coagulopathy is present (see King's College Criteria).

Nonsteroidal anti-inflammatory drugs (NSAIDs)

Although individual analgesics rarely induce liver damage due to their widespread use, NSAIDs have emerged as a major group of drugs exhibiting hepatotoxicity. Both dose-dependent and idiosyncratic reactions have been documented. Aspirin and phenylbutazone are associated with intrinsic hepatotoxicity; idiosyncratic reaction has been associated with ibuprofen, sulindac, phenylbutazone, piroxicam, diclofenac and indomethacin.

Glucocorticoids

Glucocorticoids are so named due to their effect on the carbohydrate mechanism. They promote glycogen storage in the liver. An enlarged liver is a rare side-effect of long-term steroid use in children.The classical effect of prolonged use both in adult and paediatric population is steatosis.

Isoniazid

Isoniazide (INH) is one of the most commonly used drugs for tuberculosis; it is associated with mild elevation of liver enzymes in up to 20% of patients and severe hepatotoxicity in 1-2% of patients.

Natural products

Examples include many amanita mushrooms (particularly the destroying angels), and aflatoxins

Industrial toxin

Examples include arseniccarbon tetrachloride, and vinyl chloride.

Herbal and alternative remedies

Examples include: Ackee fruitBajiaolianCamphorCopaltraCycasinKava leaves, pyrrolizidine alkaloidsHorse chestnut leaves, ValerianComfrey. Chinese herbal remedies: Jin Bu HuanMa-huangShou Wu PianBai Xian Pi.

Take dietary supplement to flush out those toxins! Empower your cells with Glutathione!

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Toxins in Cosmetics


Take dietary supplements to help flush out toxins inside your body. Empower your cells!

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Glutathione on people suffering with ALS


Antioxidants for ALS - Most of you have heard of antioxidants and that they are good for you.Antioxidants are substances that neutralize free radicals. Free radicals are highly reactive chemicals produced in our bodies that indiscriminately attack and destroy tissue causing aging, inflammation and disease.

Oxidative stress is a condition that arises when our cells cannot adequately destroy the free radicals we make. Oxidative stress results primarily from environmental toxins such as toxic metals and chemicals, alcohol, smoke, smog, prescription drugs, emotional stress, too much exercise and a poor diet. Since we are exposed to more environmental toxins than ever before, degenerative diseases are appearing earlier, more often, and to a much more severe degree.


Free radical damage is associated with many degenerative disorders including ALS. Neurological disorders create a massive production of free radicals over a long period of time and a depletion of the body's antioxidant defenses.

In a study published by Neurochemical Research, ALS patients had significantly higher levels of oxidative stress compared to controls. And virtually every team of scientists investigating ALS world-wide agree that the free radicals play a central role in the destruction of brain neurons.


Protect Yourself with Antioxidants for ALS

Reducing free radical damage in the body is critical for all patients with neurological disorders. Antioxidants combat free radicals by providing protection from oxidative stress. Several studies have shown that increasing antioxidants in the diet slows the course of neuro-degenerative disorders and may prevent these diseases in some cases.

The most important antioxidants include detoxification enzymes such as glutathione, vitamins such as vitamin C and vitamin E, and nutritional supplements such as coenzyme Q10.

Coenzyme Q10
Coenzyme Q10 (CoQ10) is a fat-soluble natural vitamin-like substance produced by the body, and serves two major functions. CoQ10 is a powerful antioxidant that protects cells against free radical damage, and is also a natural compound that acts inside the mitochondria, the "powerhouse" of the cell, where it's involved in cell energy production.

Researchers have found that people with severly degenerative neurological conditions have low levels of CoQ10 in their bodies. There is some preliminary evidence from neurodegenerative animal studies that CoQ10 may be beneficial for People with ALS:

1. In an ALS mouse model, oral CoQ10 significantly prolonged the lives of mice with mutated SOD1 genes. The CoQ10 was found to penetrate into brain tissue in general and into the mitochondria of brain cells. The researchers concluded that "CoQ10 can exert neuroprotective effects that might be useful in the treatment of neurodegenerative diseases, such as Huntington's and ALS."

2. In human clinical trials, CoQ10 slowed the progression of Parkinson's disease and generated less functional decline in Huntington's disease. Both Parkinson's disease and Huntington's disease are neurodegenerative diseases like ALS, and share similar biological pathways.

According to David Perlmutter, M.D., a Board Certified Neurologist, "Coenzyme Q10 dramatically extends survival in animal models of ALS. So there is no reason to wait for clinical research to prove CoQ10's effectiveness in humans. Time is clearly of the essence in ALS. Including coenzyme Q10 in this protocol is mandatory."

Based on this very preliminary research, you may want to take 100 mgs of an oil-based CoQ10 supplement 1 to 3 times a day. CoQ10 has been found to be safe and relatively well-tolerated, even at relatively high doses.

How to Choose a Good CoQ10 Supplement

Coenzyme-Q10 (or CoQ10) is available from many manufacturers in a number of different forms.

Several manufactureres have worked on developing enhanced bioavailability forms of Coenzyme Q10, meaning that if more of the CoQ10 is absorbed by the body, then a lower dose of the supplement is needed. The gel form of reduced CoQ10 is called ubiquinol, which is far superior to the more common form of CoQ10 called ubiquinone, which has been oxidized.
Until recently the reduced form ubiquinol was difficult to manufacture due to its rapid oxidation in the air. New technology has allowed for the manufacturing of stable coenzyme Q10 in the reduced form. The results from an animal study that compared the absorption of the two different forms, as well as a human study showing significant absorption of ubiquinol, showed that taking ubiquinol orally will offer much improved bioavailablility.

NOTE: Although CoQ10 is safe and relatively well tolerated, roughly two percent of individuals using CoQ10 experience side effects that may include nausea, upset stomach, rash, diarrhea, headache and irritability. Although there is no concrete published data on CoQ10's interactions with other drugs, some information suggests that CoQ10 may interact with certain blood thinners, such as Warfarin (Coumadin).

Glutathione
Glutathione is one of the most crucial and powerful antioxidants naturally present in the body. It has many important funtions, two of them critical for patients with neurological disorders. ALS patients have been found to have strikingly low levels of glutathione in their cerebrospinal fluid, even when other biochemical changes are not yet detectable. This has profound implications - not only may a deficiency of glutathione play a role in the development of ALS, but its supplementation may be a powerful therapeutic intervention.

Glutathione's potential is supported by research from Harvard Medical School. The authors stated that, "Pertubations for free radical homeostasis are proposed to cause ALS. A corollary hypothesis is that antioxidants should slow the course of the disease. One strategy for antioxidant therapy is to manipulate levels of glutathione...". What they are saying is that supplying the body with glutathione may be able to favorably affect the healing process for neurological patients.


Glutathione's Two Critical Functions
1) It is a major player in detoxifying the body of many toxic pollutants, including toxic metals and chemicals. Glutathione deficiency impairs the body's ability to get rid of toxins whether they are environmental or the by-products of cellular metabolism. If we have low glutathione levels we slowly become toxic, storing away poisons in our tissues, organs, muscles and brain. We simply cannot detoxify effectively if our glutathione levels are too low, no matter what form of detoxification we undertake.

For instance, epidemiological studies have shown that ALS is twice as common in individuals who have a history of exposure to agricultural chemicals, and twice as common in Gulf War veterans. However, not all exposed individuals go on to develop ALS. The reason may be that certain people lack the ability to eliminate toxins because of inadequacies in their detoxification systems. Glutathione deficiency is directly linked to the reduced ability to detoxify.

2) Glutathione is a powerful antiviral and antimicrobial agent. A glutathione deficiency compromises our ability to fight off bacteria and keep old viruses dormant. This is why so many of us test positive for Epstein Barr Virus, Cytomegalo Virus, Mycoplasma, Chlamydia Pneumoniae, Poliomyelitis, etc. ALS researchers have long suspected a viral component to ALS, since many neurodegenerative disorders are associated with chronic infections, including mycoplasma and latent virus infections.

According to David Perlumetter, M.D. (a board certified neurologist):
“Intriguing research published in 1999 by Japanese scientists has revealed a striking deficiency of this brain-protecting chemical (glutathione) in the cerebrospinal fluid of patients diagnosed with ALS…Not only may glutathione deficiency play a role in the genesis of ALS, but its repletion offers the potential for a powerful therapeutic intervention”.

Source: http://www.evenbetterhealth.com/als-antioxidants.php

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What is Alzheimer's Disease?

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Alzheimer's disease is a progressive, degenerative neurological condition caused by nerve cell death resulting in atrophy of the brain. 

Alzheimer's is the most common form of dementia,which is predicted to become the number one health issue in Australia this century. The incidence of Alzheimer’s disease is increasing. It is thought that three percent of people aged 65- 74 have the disease and those over 85 have a higher risk closer to fifty percent.

Symptoms of Alzheimer's Disease

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Alzheimer's disease is defined as "an irreversible, progressive brain disease that slowly destroys memory and thinking skills, eventually even the ability to carry out the simplest tasks”.

Early symptoms of the disease, often mistakenly attributed to aging or stress, include irritability, aggression and mood swings. There can also be subtle problems with planning, attentiveness, flexibility, apathy and memory impairment. These early mild symptoms may present many years before the clinical signs of disease are evident. As the disease advances however, significant confusion, language difficulties and memory loss occurs.

What causes Alzheimer's Disease?

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Scientists do not yet fully understand the causes of Alzheimer’s disease (except for the 1 – 5% of cases which have a genetic basis).

There is growing evidence that lifetime exposures to toxins, chemicals, poor diet and oxidative stress are all factors, triggering inflammatory processes and a build up of beta amyloid plaque in the brain cells’ mitochondria.

Glutathione depletion is known to be a key factor in Alzheimer’s Disease.

Electromagnetic Radiation

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Scientific studies have shown that exposure to electromagnetic radiation significantly increases the risk of Alzheimer’s, or can make it rapidly worse. Wi-fi, cordless telephones and mobile phones all create microwave radiation which has been shown in scientific studies to cause genetic mutations in brain cells. These mutations may lead to dementia or cancer.

Heavy Metal Toxicity

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Another part of the puzzle is exposure to toxic heavy metals. Aluminium was originally thought to be the main offender although today we realise that mercury poisoning seems a far more likely culprit. We are exposed to very high levels of mercury from dental amalgam and various dietary sources, such as fish. Chelation can remove these toxic metals from the tissues.
Some doctors have recognized the value of chelation for Alzheimer's and other degenerative disorders caused by metal poisoning and have found it to be very effective at restoring the function of older blood vessels and brain cells. Glutathione is able to chelate these toxic metals. 

Possible Natural Treatments

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No effective medical treatment is available for this debilitating disease, although mental stimulation, exercise and a balanced diet are all thought to help delay symptoms. Antioxidants such as Vitamin E, B6, B12, folate, and Gingko biloba are thought to be of some benefit. Glutathione is our body's own most powerful antioxidant and has an important role in the brain.

Glutathione helps fight oxidative stress and can have remarkable effects on mental performance and cognition. It is very helpful for Alzheimer’s patients to supplement their glutathione levels.

The Glutathione Connection

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Loss of intracellular neuronal glutathione (GSH) is an important feature of neurodegenerative disorders including Alzheimer's disease, Parkinson's disease, and amyotrophic lateral sclerosis. The consequences of GSH depletion include increased oxidative damage to proteins, lipids, and DNA and subsequent cytotoxic effects. 
(ref Du et al, Free Radical Biology and Medicine 2008)

Glutathione deficiency contributes to oxidative stress, which plays a key role in aging and the pathogenesis of many diseases, including Alzheimer's and Parkinson's disease. Scientists have demonstrated that supplementing with glutathione precursors can product beneficial effects in slowing the progression of these diseases.

Dr David Perlmutter observed what he called the "glutathione miracle" when he gave patients with severe Parkinson's disease intravenous glutathione. Within one hour they were able to stand, walk and move their arms around again normally. He has also used glutathione extensively in cases with Alzheimer's and other dementias, multiple sclerosis, strokes and chronic fatigue with success.


How to Boost Glutathione Levels






Glutathione must be synthesised within the cells  because it cannot be transported into cells. Thus, taking glutathione orally will not work as it is simply broken down in the stomach. The novel compound RiboCeine™ is able to effectively solve this problem and deliver the essential precursor nutrients to the cells where they can then be turned into glutathione.  

RiboCeine is far more effective than any other form of glutathione enhancement and is uniquely found in the supplement Cellgevity

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Glutathione is Deficient in the Brains of Patients with ADHD

Glutathione is Deficient in the Brains of Patients with ADHD

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Scientific studies have found evidence of significant glutathione depletion in patients who have symptoms of Autistic Spectrum Disorder and Attention Deficit Hyperactivity Disorder (ADD or ADHD)

Glutathione is the body's master antioxidant and is vital for counteracting oxidative stress. When a brain neurological disorder is present, glutathione levels become depleted, leading to a build up of cell damage caused by free radicals and toxins from the environment. Glutathione levels are also further depleted by common medications such as acetaminophen (paracetamol).

Electromagnetic Radiation (EMR)

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Glutathione also helps to protect our cells from damage caused by radiation. One form of radiation that the brain is acutely sensitive to is electromagnetic radiation (EMR). 
Scientists have reported that microwave radiation from wireless devices (such as cordless phones, cellphones and WiFi) causes the blood brain barrier to become permeable, allowing more toxins to enter the brain.   Read more

The Importance of a Healthy Diet

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A healthy diet is essential in maintaining the right balance of nutrients in the brain. Certain foods such as asparagus, broccoli, watermelon are ideal for supporting healthy brain function. Fresh fish also provides an important source of Omega 3 fatty acids, which are another vital component of brain health. Processed foods and sugary drinks should be avoided. 

However in this modern world where soils are often depleted of nutrients and foods are laden with toxic pesticides, a balanced diet alone is often not enough to maintain healthy glutathione levels. 

RiboCeine™ increases Glutathione levels

MaxOne and Cellgevity
C

Glutathione is a tripeptide molecule that is made within every cell of the body from three amino acids: glutamate, glycine, and cysteine. Glutathione (GSH) cannot be supplemented orally as the fragile molecule is simply broken down in the digestive tract. Instead the necessary precursors (building blocks) must be provided in a form that the body can absorb. 

The novel compound RiboCeine™ provides these precursors in a highly bioavailable form, allowing the cells to manufacture glutathione. 

MaxOne™ and Cellgevity™ are advanced glutathione supplements powered by RiboCeine™, clinically proven to significantly increase glutathione production throughout the body.
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